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    淡江大學機構典藏 > 體育事務處 > 專書 >  Item 987654321/110403


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    题名: 低加壓高阻力運動對肌肉增生生化指標及運動表現之影響
    其它题名: The effect of low vascular occlusion combined with high-intensity resistance exercise on muscle hyperplasia and inflammatory markers and exercise performance
    作者: 何松諺
    关键词: 肌肉損傷;muscle damage;最大肌力;maximal strength;加壓訓練;occlusion
    日期: 201206
    上传时间: 2017-06-17 02:10:14 (UTC+8)
    摘要: 目的:探討加壓阻力訓練對角力運動員肌肉量、肌肉增生激素、肌肉發炎指標、最大肌力及左右敏捷性之影響。研究方法: 14名角力專項運動員(年齡:20.4 ± 1.3歲;身高:169.6 ± 6.4公分;體重:76.8 ± 17.3公斤),進行3週加壓阻力運動訓練,每週進行3天,一天5組,每組12次,組間休息1分鐘,運動強度為70% 1RM,加壓壓力以每位受試者的0.7倍收縮壓,於運動前、中、後採取肌肉增生激素 (IGF-1),並於運動前及每週運動後採集肌肉發炎指標。結果:無論是否有加壓的介入,受試者之肌肉量均有顯著增加之情形 (加壓前測:9.5 ± 1.27,加壓後測9.6 ± 1.25:未加壓前測:9.5±1.33 未加壓後測:9.7±1.36 kg, p<.05);肌肉增生激素IGF-1於訓練前、中、後皆無顯著差異(p>.05);肌肉發炎指標方面,CK及GOT皆無顯著差異 (P>.05),CK-MB第二週及第三週之活性顯著高於前測 (前測:6.5 ± 4.53,第一週:10.2 ± 11.09,第二週:11.6 ± 1.94,第三週:11.2 ± 3.28 U/L, p<.05),GPT第二週活性顯著高於第三週 (前測:9.8 ± 6.43,第一週:12.6 ± 7.71,第二週:17.5 ± 10.05,第三週:11.2 ± 5.40 U/L, p<.05),而LDH第一週之活性則顯著高於第二週 (前測:164.2 ± 29.30,第一週:190.4 ± 54.19,第二週:152.9 ± 18.60,第三週:11.2 ± 3.28 U/L, p<.05);最大等長收縮及最大等速收縮之肌力,於運動訓練後皆顯著低於運動訓練前 (p<.05)。另外,三週的實驗處理後,受試者之乳酸閾值有顯著增加的情形 (前測:36.9 ± 10.43,後測:45.5 ± 7.80 ml∙kg-1 ∙min-1, p<.05),而最大攝氧量有些微的降低,但在統計上則無差異。結論:三週加壓阻力運動訓練對肌肉增生指標沒有影響,最大肌力值呈現下降的情形。另外,受試者的乳酸閾值有顯著改善,且腿部肌肉量有顯著之進步。
    Purpose: The aim of this study was to investigate the effect of resistance exercise combined occlusion training on wrestling athletes’ muscle mass, muscle hyperplasia, inflammatory markers, maximal voluntary contraction and agility test. Method: Fourteen wrestling athletes (age:20.4 ± 1.3yrs;height:169.6 ± 6.4cm;weight:76.8 ± 17.3kg) were recruited in this study to perform bilateral leg extension for 3 weeks, 3 days per week and 5 sets, 12 repetitions with 1minute rest among all sets each day. The intensity was 70% 1RM with low occlusion pressure (0.7SBP). The muscle hyperplasia blood sample were collected prior to, during and after exercise. The inflammatory markers were collected before and every week after training. Result:No matter occlusion or not, a significant increased in the muscle mass (occlusion-pre:9.5 ± 1.27, occlusion-post:9.6 ± 1.25, No occlusion-pre:9.5±1.33, No occlusion-post:9.7 ± 1.36 kg, p<.05).There was no significant difference in IGF-1 in the test (p>.05). The inflammatory marker of CK and GOT were the same result (p>.05). CK-MB was higher at second and third week than pre (pre:6.5 ± 4.53,week-1:10.2 ± 11.09,week-2:11.6 ± 1.94,week-3:11.2 ± 3.28 U/L, p<.05). GPT was higher at second week than third week (pre:9.8 ± 6.43,week-1:12.6 ± 7.71,week-2:17.5 ± 10.05,week-3:11.2 ± 5.40 U/L, p<.05), and LDH was higher at first week than second week(pre:164.2 ± 29.30,week-1:190.4 ± 54.19,week-2:152.9 ± 18.60,week-3:11.2 ± 3.28 U/L, p<.05). The maximal voluntary contraction of isokinetic and isometric were lower after occlusion training (p<.05). In addition, the lactate threshold was higher after the training (pre:36.9 ± 10.43,post:45.5 ± 7.80 ml∙kg-1 ∙min-1, p<.05) and the VO2max was no significant difference (p>.05). Conclusion: The mode of low occlusion pressure combined with high intensity resistance exercise induce higher in lactate threshold and muscle mass, but the maximal voluntary contraction was lower after training. And the muscle hyperplasia was no change.
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